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elevated cholesterol levels PART 1
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elevated cholesterol levels PART 1

the link between
elevated cholesterol levels — even LDL-cholesterol
— and heart disease, is much weaker than
measures of inflammation — especially highly
sensitive CRP (hsCRP).
The latest studies show that inflammation is an
independent risk factor for heart disease that is
much stronger than any measure of cholesterol.1
I cite a study of two statin drugs, atorvastatin and
pravastatin, that was reported in the 2005 issue of
The New England Journal of Medicine.2
The study found that patients with low hsCRP
levels had fewer heart attacks no matter their LDLcholesterol
level, and they had more heart attacks if
their hsCRP was elevated regardless of their LDLcholesterol
level.
The same thing has been found for stroke
risk. Another recent study, the Pravastatin or
Atorvastatin Evaluation and Infection Therapy —
Thrombolysis in Myocardial Infarction 22 (PROVE
IT-TIMI 22) study, examined patients on high and
moderate doses of statin drugs.
The study found that in both groups, there was
no difference in the cholesterol levels of those with
cerebrovascular event (stroke) and those without.
The only difference was the levels of hsCRP — that
is, inflammation.
The Inflammation Link
Few physicians who prescribe statin drugs know
that the link between elevated cholesterol levels and
strokes has never been established, but the link to
inflammation is strong and is supported by many
laboratory and clinical studies.3
Reported reductions in stroke risk for people
taking statins have varied from no statistical
reductions (as in the Treating to New Targets or
TNT study) to 19 percent to 50 percent shown
in the Long-term Intervention with Pravastatin
in Ischaemic Disease (LIPID), The Cholesterol
and Recurrent Events (CARE), and Myocardial
Ischemia Reduction with Aggressive Cholesterol
Lowering (MIRACL) studies.
Newer evidence, however, suggests that any
reduction in stroke risk is secondary to the antiinflammatory
effects of the drugs instead of their
ability to lower cholesterol.
Why is there so much deception? Why won’t the drug manufacturers and physicians who promote
statin drugs just change their policy and give statin
drugs only to people with increased inflammation?
It all comes down to dollars.
If I make lowering cholesterol my goal —
especially a drastic decrease — I can convince
doctors that everyone, even children, should take
them for a lifetime.
Yet, if I use hsCRP or other measures of
inflammation as the criteria for prescribing statins,
the drug would not be given to 75 percent to 80
percent of people presently prescribed to them.
That is a massive loss of revenue.
The same situation applies with vaccines.
If you can convince most doctors that vaccines
are critical for public health, and better yet, have
states pass mandatory vaccine laws, your profits
increase enormously.
Here is an analogy: What if the government
stated one day that everyone must use Detergent A
to wash clothes and that everyone’s clothes should
be washed every day. That would mean a lot of
money for the makers of Detergent A.
Then the experts determine that finding lint in
the lint filter is linked to the need to clean clothes
— after all, we find lint every time we dry clothes.
Still later, they mandate that you should wash your
clothes with Detergent A every day even if they are
clean.
Alternatives to Statin Drugs
Now that the statin promoters have admitted
that inflammation, not elevated cholesterol, is the
cause of atherosclerosis, we can examine ways to
reduce inflammation in our bodies (and our blood
vessels in particular) rather than using statin drugs.
First, we will examine why certain people are
chronically inflamed. There are a great number of
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