In layman's terms what does this report mean for my mum who is 83 and can't use her leg because it is mostly numb.
There Is extensive FLAIR abnormality Involving the deep white matter of both cerebral hemispheres, with prominent perivaSCUlar spaces and no restricted diffusion, The appearances ara Consistent with chronic small vessel Ischemic change. There is no blooming artefact to Indicate a haemorrhage or amyloid angiopathy. No restricted diffusion is seen to Indicate recent Infarction, The ventricular system Is normal in configuration. Normal vascular flow voids demonstrated. The pituitary gland Is atrophic, with a thin rim of residual gland evident In the floor of the sella. No mass lesion Is seen.
The cord is normal in signal and calibre, and ends at a normal level of L1. No lesions are seen in the cord or intradural spaces. The L1 vertebral body demonstrates wedging and 1088 of height Involving the superior end plate centrally, in keeping with an osteoporotic insufficiency fracture. Degenerative changes at other levels, with sclerosis.
At the level of the C5/6 Intervertebral disc. there Is a broad-based disc protrusion which is effacing the thecal sac. The disc has lost height, and there are coarse osteophytes posteriorly. This is producing some canal stenosis, particularly laterally, though the cord at this level is of normal signal and calibre. Similar changes at can disc level. though with a lesser degree of canal stenosis. These changes are also producing foraminal stenosis at the C5/6 and C6/7 levels. Broad-based protrusions are also seen at the L213, L4/5 and L5/S1 discs, but there Is no evidence of foraminal or central canal stenosis.
COMMENT: Extensive chronic small vessel ischemic change in both cerebral hemispheres. No evidence of recent cortical Infarction, and no haemorrhage evident.
Multi-level posterior disc protrusions, at the level of the C5/6 particularly, is causing some spinal canal stenosis, though the cord is of a normal signal, and therefore clinical relevance Is doubtful.
Insufficiency fracture of L1 without evidence of canal stenosis.
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