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2d echo interpretation

2d echo interpretation

hi! just want to know if i should seek further test on my heart because of the recent echo findings. the findings was "there is a concentric left ventricular hypertrophy with good wall motion and contractility and preserved systolic function" my ef=61%. I have a blood pressure problem taking metoprolol 100mg and also i have elevated heartrate during mornings at rest ranging from 100 to 120bpms for around an hour. thanks!  
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If you are already taking 100mg metoprolol and you are still having spells of tachycardia, then yes, do continue to seek help.  One would hope that the metoprolol would control your symptoms, and if not, then further consultation is appropriate.  
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367994_tn?1304957193
To give you some insight concentric LV hypertrophy.  With sustained hypertension the heart develops concentric hypertrophy that is characterized by thickening of the intraventricular septum (wall that separates left and right chambers) and the contracting wall of the left ventricle.

Your therapy is timely as the hypertrophic process is initially adaptive, in that it enables the heart to maintain normal pump performance (systole) in the face of the increased afterload (high blood pressure). Without proper treatment there is an  increase in fibrillar collagen that prevents the heart from contracting and relaxing normally.

You have to get your blood pressure under control, and the increase of the periodic heart rate should be diagnosed  Your EF of 61% indicates your heart's contractions are  adequate...normal EF is 50 to 70%, and it indicates the amount of blood pumped into circulation with each heartbeat.
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thanks a lot, you're explanation enlightens me, thanks again. do you have any idea about intraventricular conduction delay? my ecg has a findings of ivcd and i dont have any idea what it is. is there any treatment on that? thanks.
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367994_tn?1304957193
Question: my ecg has a findings of ivcd and i dont have any idea what it is. is there any treatment on that? thanks

>>>The ECG appearance of IVCD (intraventricular condition delay) is difficult to characterize.  It can be the end result of a number of different pathophysiologic processes. Examples of conditions that may lead to IVCD include myocardial infarction, cardiomyopathy with  ventricular fibrosis, chamber enlargement.

The QRS complex is wide (QRS measures mvolts and msec of electrical impulse as it passes through the left ventricle), but neither typical RBBB nor typical LBBB is present (assuming no bundle branch block which is an interference along the impulse pathway).  In this case, the reason for QRS widening must be the presence of IVCD (IntraVentricular Conduction Delay).  For some insight the delay of conductivity of an impulse is due to the higher resistance of scar tissue from an MI,  also high resistance to pass an impulse with ventricular fibrosis, and an enlarged chamber lengthens (delays the impulse).
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