My electrophysiologist thinks I either have av nodal reentry
or pat from the heart monitor. He's not sure which.
What is the difference and is one worse then the other?
Everything I've read said this is not life threatening.
He tried three medicines and I am currently on
atenololAtenolol
Atenolol-chlorthalidone 25 mg
once a day. None of the medicines seem to get rid of this
problem. I just don't feel it as bad. It's also not as long.
Usually if I take a deep
breathBreath alcohol test
Breath holding spell
Breath odor and hold it a couple of times
it will go back into a
normalNormal saline flush rythem. One time it felt like
my heart stopped and I almost fainted but since my palse is so
weak with the medicine I couldn't find it to see what was wrong.
Just when I thought I was gonna
blackBlack cohosh
Black draught
Black haw out my heart started beating
again. It probably wasn't as long as it felt but it was scary.
My heart still does the same
rapidRapid shallow breathing heart beat that it was doing without
medicine. My last visit he recommended increasing the
atenololAtenolol
Atenolol-chlorthalidone to 35 mg a day but I wanted to wait. Why doesn't
the medicine work? He said he could go in a try to see
if he could zap it but wasn't ready for that either. Is this serious or not?
Here is an explanation I found on another site about how they differ:
"Tachycardias involving the AV node:
AV nodal reentrant tachycardia. An arrhythmia due to an extra conducting pathway within the AV node. This allows the heart’s electrical activity to “short circuit” itself (“reentry”). Episodes of this arrhythmia may be triggered by physical or emotional stress, caffeine or certain medications. AV nodal reentry can often be managed by medical therapy with beta blocker or calcium channel blocker medications, but can also be cured by catheter ablation of the extra pathway.
AV reentrant tachycardia using an accessory bypass connection. Similar to AV node reentry, this occurs when an extra conducting pathway allows the electrical impulse to “short circuit.” In contrast to AV node reentry, however, the extra pathway in this condition bypasses the AV node, directly linking the atria and ventricles. In most cases, this pathway can only conduct “backwards” — from ventricles to atria. This is called a “concealed accessory pathway” since it cannot be diagnosed from a regular electrocardiogram (EKG). These arrhythmias may be treated medically, but can also be cured by catheter ablation. Less often, the extra pathway is evident on the EKG, in which case the condition is called the Wolff-Parkinson-White syndrome (WPW). WPW syndrome may result in extremely rapid heartbeats and could potentially result in death. Symptomatic WPW syndrome generally requires catheter ablation."
"Atrial tachycardias may be focal (arising from only one place in one of the atria) or multifocal (arising from many different places in the atria).
Paroxysmal atrial tachycardia. A condition originating in the atria, in which the heartbeat increases for several minutes to a number of hours. Treatment may be necessary for short, sudden episodes that begin and end rapidly. Most people with this condition are young with normal hearts. The condition may be exacerbated by the use of even small amounts of caffeine or alcohol. This condition has also been associated with overly high levels of digitalis (a cardiotonic drug) in the bloodstream......"
I am not on medication and am on the list for the RF ablation procedure.
Marie