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Blood pressure rises while sleeping
I am 46 yrs. old. I have high blood pressure. My doctor has changed my bp medicine twice & next week I am to see a cardiologist. My blood pressure rises while I am asleep & at times wakes me up because I have fast palpitations in my heart. I get up & check my bp & it ranges from 165 - 190 to 110 - 128. So basically the minimum is 165/110. During the day it ranges from 140/96 - 160/108. Basically I have hard time getting back to sleep because my heart is racing. I also have asthma. I have tried finding on the computer why my bp rises while sleeping & really can't find answers. It usually say's bp lowers while sleeping & mine rises while sleeping. I wake up a few times a night. I am calm when I go to bed at nights. I don't have anxiety. It just scares me how high my bp gets while I am sleeping. Any advice. Should I be concerned or is it okay for me to go the cardiologist a week from tomorrow. I worry I may have a heart attack or some kind of heart failure while asleep. I would say 80% of the time my blood pressue rises while I am asleep.
I awake and my blood pressure is 179 over 99 and an hour later it’s 120 over 70. Why
Just do deep breathing for 10 minutes then do a blood pressure it will go down the reason the blood pressure is rising because you’re not getting enough oxygen to your brain so the brain send a signal to the kidney to the raise the blood pressure by lowering the blood pressure it will cause heart attack stroke and other problems you just have to deep breathe for 10 minutes a day your blood pressure will go down thank you
I have been waking up with HPB 210/130/48 some times. No troubles while I was being treated for cancer, now the hip is returning. More meds, I feel my issue is mental, but I wonder because of the rapid rise in HPB and the excellarated drop after. My mother died of strokes, my grand mother the same, my brother is an invalid at this time for the same reason. I purchased a Fitbit to monitor, heart rate while sleeping 40-109. I lost my left ear drum, so I hear my hear and by now I can tell different bP and heart rate just by listening. Yogo helps bring it down. I can meditate and drop 50-90 points on the upper end and 10-30 on the lower end. I think may indicate stress. Any thoughts?
ashwaganda and chamomile tea did not help me. I have the same as you, also have developed tinnitus and hearing my heart pound in my ears. One ear feels like its full of water but isnt. Had catscan, all clear. I have not found anything that helps, and the spiking is getting worse and the heart racing waking me episodes are almost every night now. Have cut excess sugar, all caffeine, I dont smoke, dont do drugs, dont drink, never have. My only vice was candy and cookies...I'm 100 pounds overweight. Working on that now, and to exercise daily. I think for me it is hormones. Even for a man, it can be lowered testosterone. Have a spit test done. I'm going tomorrow to ask for diabetes, thyroid, spit test for hormones, and if that shows nothing, I'll ask to have my adrenal gland checked for tumor. Heart is normal. If I can't figure this out, I'm screwed.......
1 Comments
Are you feeling better now? My mom has the same issue . Blood pressure rises while sleeping .. Yesterday she got 128/85 before sleep, then she woke up 2 hours later with 152/95 , then at morning 6:00 am 175/112 .. She is taking her medicine at morning and before sleep , but at night seems like there is no results .. Through the day she feels good, blood pressure around 128/80 . I would appreciate if you answer me
QUOTE: I have tried finding on the computer why my bp rises while sleeping & really can't find answers.
Does the following information add to what you have have learned from the internet?
Mayo Clinic:Having an abnormal blood pressure pattern, such as high blood pressure in the morning, can mean that you have a health problem. Potential problems include:
■Poorly controlled high blood pressure
■Sleep apnea
■Tumors of the adrenal gland
■Kidney disease
Obstructive sleep apnea: one hypoxic (decrease of oxygen level in the blood/decreased oxygen to heart cells) night increases daytime blood pressure. Also, asthma decreases oxygen level in the blood and can contribute to higher than normal bp.
Severe intermittent hypoxia, such as that experienced during episodes of obstructive sleep apnoea, results in a maintained elevation in blood pressure and a sympathetic hyperactivity, French researchers report in the European Respiratory Journal.
Obstructive sleep apnea syndrome (OSAS) is associated with nocturnal chronic intermittent hypoxia that leads to excess cardiovascular morbidity. Some of the proposed contributing mechanisms are an increased vascular sympathetic tone, resulting in an elevated systemic resistance and hypertension, an impaired arterial vasodilatory capacity, and chronic inflammation leading to endothelial dysfunction and atherosclerosis.
In order to better understand the pathophysiological events triggered by intermittent nocturnal hypoxia, Dr Tamisier and colleagues of the Laboratoire d'Exploration Fonctionnelle Respiratoire at the University Hospital in Grenoble examined whether the effect of intermittent nocturnal hypoxia on arterial blood pressure is sustained throughout the waking hours and whether it extends beyond the acute episodes.
The authors tested if, in young and healthy subjects, after 2 weeks of hypoxic attacks with 30 desaturations per hour, arterial blood pressure would remain elevated and if this would relate to increased sympathetic activity to muscle vasculature, decreased flow-mediated dilatation and increased circulating biomarkers of inflammation. They performed 24-h ambulatory monitoring of blood pressure in 12 healthy subjects before and after 2 weeks of exposure to intermittent hypoxia. They also assessed haemodynamic parameters, muscle sympathetic nerve activity, the baroreflex control of sympathetic response and inflammatory markers in the blood before, during and after exposure.
After a single night of intermittent hypoxia, mean blood pressure during the day increased by 3 mmHg and further increased after 2 weeks of exposure (8 mmHg systolic and 5 mmHg diastolic). Mean sympathetic nerve activity to the muscles increased across exposure time and the baroreflex control of sympathetic outflow declined. There were no evident changes in either vascular reactivity or systemic inflammatory markers.
Although intermittent hypoxia was applied during sleep, increased blood pressure occurred during the daytime and not the night-time, the authors report. The rise in blood pressure was sustained throughout the daytime, beyond the acute phase of severe intermittent hypoxia, but returned to baseline after 5 days of recovery.
These data are the first to show that increased sympathetic activation induced by intermittent hypoxia probably contributes to blood pressure elevation and may derive from a decreased baroreflex function. These mechanisms may reflect those underlying the blood pressure elevation associated with OSAS.
However, one cannot extrapolate from these findings in young, healthy individuals to the typical (overweight and middle-aged) obstructive sleep apnoea patients. In this very young and lean cohort, intermittent hypoxia did not lead to any increase of nocturnal catecholamine excretion. Moreover, the employed model of hypoxia itself is not comparable to OSAS: a patient with OSAS experiences asphyxia during sleep, i.e. he suffers from both hypoxia and hypercapnia, whereas the experimental model reduces oxygen as well as carbon dioxide. This may lead to an underestimation of the effect of hypoxia, since increased carbon dioxide enhances the cardiovascular responses to hypoxia in both healthy individuals and sleep apnoea patients, the authors caution.
Reference:
14 nights of intermittent hypoxia elevate daytime blood pressure and sympathetic activity in healthy humans.
Tamisier R et al. Eur Respir J. 2011; 37: /119 N 128.
Thanks for sharing and if you have any other questions or comments you are welcome to respond. Take care and I wish you well going forward.
Ken
Does the following information add to what you have have learned from the internet?
Mayo Clinic:Having an abnormal blood pressure pattern, such as high blood pressure in the morning, can mean that you have a health problem. Potential problems include:
■Poorly controlled high blood pressure
■Sleep apnea
■Tumors of the adrenal gland
■Kidney disease
Obstructive sleep apnea: one hypoxic (decrease of oxygen level in the blood/decreased oxygen to heart cells) night increases daytime blood pressure. Also, asthma decreases oxygen level in the blood and can contribute to higher than normal bp.
Severe intermittent hypoxia, such as that experienced during episodes of obstructive sleep apnoea, results in a maintained elevation in blood pressure and a sympathetic hyperactivity, French researchers report in the European Respiratory Journal.
Obstructive sleep apnea syndrome (OSAS) is associated with nocturnal chronic intermittent hypoxia that leads to excess cardiovascular morbidity. Some of the proposed contributing mechanisms are an increased vascular sympathetic tone, resulting in an elevated systemic resistance and hypertension, an impaired arterial vasodilatory capacity, and chronic inflammation leading to endothelial dysfunction and atherosclerosis.
In order to better understand the pathophysiological events triggered by intermittent nocturnal hypoxia, Dr Tamisier and colleagues of the Laboratoire d'Exploration Fonctionnelle Respiratoire at the University Hospital in Grenoble examined whether the effect of intermittent nocturnal hypoxia on arterial blood pressure is sustained throughout the waking hours and whether it extends beyond the acute episodes.
The authors tested if, in young and healthy subjects, after 2 weeks of hypoxic attacks with 30 desaturations per hour, arterial blood pressure would remain elevated and if this would relate to increased sympathetic activity to muscle vasculature, decreased flow-mediated dilatation and increased circulating biomarkers of inflammation. They performed 24-h ambulatory monitoring of blood pressure in 12 healthy subjects before and after 2 weeks of exposure to intermittent hypoxia. They also assessed haemodynamic parameters, muscle sympathetic nerve activity, the baroreflex control of sympathetic response and inflammatory markers in the blood before, during and after exposure.
After a single night of intermittent hypoxia, mean blood pressure during the day increased by 3 mmHg and further increased after 2 weeks of exposure (8 mmHg systolic and 5 mmHg diastolic). Mean sympathetic nerve activity to the muscles increased across exposure time and the baroreflex control of sympathetic outflow declined. There were no evident changes in either vascular reactivity or systemic inflammatory markers.
Although intermittent hypoxia was applied during sleep, increased blood pressure occurred during the daytime and not the night-time, the authors report. The rise in blood pressure was sustained throughout the daytime, beyond the acute phase of severe intermittent hypoxia, but returned to baseline after 5 days of recovery.
These data are the first to show that increased sympathetic activation induced by intermittent hypoxia probably contributes to blood pressure elevation and may derive from a decreased baroreflex function. These mechanisms may reflect those underlying the blood pressure elevation associated with OSAS.
However, one cannot extrapolate from these findings in young, healthy individuals to the typical (overweight and middle-aged) obstructive sleep apnoea patients. In this very young and lean cohort, intermittent hypoxia did not lead to any increase of nocturnal catecholamine excretion. Moreover, the employed model of hypoxia itself is not comparable to OSAS: a patient with OSAS experiences asphyxia during sleep, i.e. he suffers from both hypoxia and hypercapnia, whereas the experimental model reduces oxygen as well as carbon dioxide. This may lead to an underestimation of the effect of hypoxia, since increased carbon dioxide enhances the cardiovascular responses to hypoxia in both healthy individuals and sleep apnoea patients, the authors caution.
Reference:
14 nights of intermittent hypoxia elevate daytime blood pressure and sympathetic activity in healthy humans.
Tamisier R et al. Eur Respir J. 2011; 37: /119 N 128.
Thanks for sharing and if you have any other questions or comments you are welcome to respond. Take care and I wish you well going forward.
Ken
I just saw a television show about a man who was going through a similar situation as yours. His blood pressure and heart rate would go up significantly when he was sleeping; it was so bad, his wife would have to take him to the hospital. The doctors didn't know why it was happening. It turned out he had obstructive sleep apnea...he would actually stop breathing for several seconds while he was sleeping and this would cause the dramatic rise in his bp and heart rate. His problem was found when he spent the night in a sleep clinic. They gave him a c-pap machine and he hasn't had the problem since. Perhaps you should look into this. If you're married, ask your spouse to watch you when you sleep to see if you stop breathing intermittently. Do you snore? That is a sign of sleep apnea. I wish you the best.
1 Comments
I have a similar problem. I am on cpap therapy and have been for several years, My problem started after I was on cpap therapy. This problem occurs shortly after going to sleep. I start dreaming and then wake up with high pb and my back aches, my legs hurt and I squirm around, get up and drink some hot milk in the living room and sit and wait for me to get back to normal bp which takes a complete hour. After that, no sleep. I think it happens during REM sleep. When I say high bp, 180/100/72 then it goes back to normal 130/69/72. Therefore I am sleep deprived as I only get a few hours during the night. I had two sleep tests which were ordered by my "sleep specialist?"and I had this sleep problem during both tests and was told that nothing irregular was seen. My AHI is usually .02 . I have had a heart monitor implanted and my heart activity shows normal during these episodes. Fortunately the severity and the frequency has let up some but it will not go away.
I am having same problem. Have been on 20mg lisinoprol for many years but doctor has just upped it to 40mg daily. I decided insted of taking it all at once, I am taking 20mg in a.m. and 20 mg about l2 hours later and it seems to keep my pressure down while sleeping I was getting up at night and taking the bp and it was l60/90, now with taking the two doses separately my sleeping bp has gone down to l30/80. I hate taking these meds and am looking into other alternatives. I am looking into an herb called Ashwaghanda and also an hibiscus tea. I am sorry I cannot report anything to you on this as I have not yet tried them...they are coming in the mail. From what I have read the problem might also be caused by adrenal gland dysfunction, or problem with blood flow through kidneys. You might want to research this before your visit to the doctor as I feel the more information you have on a medical visit the more will be considered. Good luck
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