EDTA chelation question

[someone21231]

I've read that the underlying cause of heart disease is free radicals damage the artery walls then calcium plugs up the hole to stop internal hemorrhaging [bleeding to death from a hole in the artery wall from free radical damage] and cholesterol sticks to the calcium. People say that EDTA chelation gets rid of arterial plague by getting rid of the calcium so wouldn't this cause death by internal hemorrhaging sometimes? This is extremely important that you answer my question. I've taken Cardio Renew a bunch of times and know it works but I'm starting to get worried that it will cause me just as bad problems as it solved not including it can screw up my kidneys and liver.

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http://www.bodyandfitness.com/Information/Health/antioxidant.htm
To much to paste here.


http://www.amazon.com/Homocysteine-Revolution-Kilmer-S-McCully/dp/0879839759/ref=cm_syf_dtl_pl_5_rdsssl0/102-1780927-4832161/102-1780927-4832161
Arterial plaque is, in essence a form of scab that forms on wounded arteries to protect us from internal hemorrhaging. What causes wounded arteries? One cause is free radicals, which can be combatted by antioxidants. But a second major cause of arterial wounding -- and arterial plaque -- was discovered by the author of this book, Dr. Kilmer McCully: elevated concentrations in the blood of a chemical called homocysteine. Homocysteine is a natural product of the metabolism af an amino acid called methionine and, therefore, our body's always contain some homocysteine. Vitamins B6, B12, and folic acid keep blood concentrations of homocysteine low enough to be safe. But deficiency of these vitamins can allow homocysteine to build up to concentrations that can wound our arteries and lead to the formation of arterial plaque -- which can eventually trigger a heart attack. Recent research has confirmed Dr. McCully's findings. If you want to understand the role of homocysteine in causing cardiovascular disease and resultant heart attacks -- and learn how to protect yourself from them -- I strongly suggest that you acquire and read this valuable book.


http://www.full-health.com/partone.htm
Atherosclerotic Plaque Formation
  
The cardiovascular system is known to be highly susceptible to free radical attack. Especially vulnerable are the endothelial cells in arterial walls. When injured, they trigger the blood platelet accumulation function, in order to protect and, eventually, heal the damaged areas.

However, if free radical damage to the endothelial cells continues, the "normal" blood platelet accumulation function becomes, at some point, abnormal and out of control - so to speak, contributing to the progressive process of arterial plaque formation, that is atherosclerosis.

The major constituents of the artery-clogging, atheromatous plaque are

    * lipids (fats)
    * foam cells (macrophages full of lipid/fat material)
    * fatty streaks, and
    * plaques in the intima of blood vessels.



http://en.wikipedia.org/wiki/Internal_hemorrhage
Platelets are small blood components that form a plug in the blood vessel wall that stops bleeding. Platelets also produce a variety of substances that stimulate the production of a blood clot.