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I am presently exercising every moring at the fitness center on the bicylcle and elliptical..,in the hopes of improving my ejecton fraction. Based on previous Q/A, is this a lost cause???
If the heart is dilated (enlarged left ventricle), the heart muscle will lose its ability to to pump adequately due to weak contractions. The phenomonon is Frank/Starling (google for more info) mechanism in physics. As an analogy when a handspring is stretched, it will spring back more forcefully, but over stretched it will become flaccid. The heart can/will compensate to increase cardiac output by dilating (maintain equalibrium of blood flow between the right and left side). But when there is heart problem and untreated, the heart will overcompensate (overworked) and there will be a loss of contractility and the EF will be lower....If a heart is the dilated cardiomyopathy classification, reducing the heart's workload can reverse remodeling and EF can/will return to normal. Medication (ACE inhibitor and beta blocker) returned my heart size to normal and my EF is currently 59%.
If the heart is dilated (enlarged left ventricle), the heart muscle will lose its ability to to pump adequately due to weak contractions. The phenomonon is Frank/Starling (google for more info) mechanism in physics. As an analogy when a handspring is stretched, it will spring back more forcefully, but over stretched it will become flaccid. The heart can/will compensate to increase cardiac output by dilating (maintain equalibrium of blood flow between the right and left side). But when there is heart problem and untreated, the heart will overcompensate (overworked) and there will be a loss of contractility and the EF will be lower....If a heart is the dilated cardiomyopathy classification, reducing the heart's workload can reverse remodeling and EF can/will return to normal. Medication (ACE inhibitor and beta blocker) returned my heart size to normal and my EF is currently 59%.