40-year old male, 6'1'' 145lb. 2005 successful ablation AVNRT. From that time experiencing new problems. The severely disabling problem that bothers me most:
Episodes start with PVC/PAC.
Instantly my heart starts beating different: unlike my
normalNormal saline flush, unnoticeable
heartbeatHeart palpitations
Ultrasound, normal fetus - heartbeat
Ultrasound, ventricular septal defect - heartbeat an extremely noticeable and forceful
heartbeatHeart palpitations
Ultrasound, normal fetus - heartbeat
Ultrasound, ventricular septal defect - heartbeat (some resemblance with slow-motion (50-70BPM) version of prior AVNRT).
Drains physical energylevel with SEVERE fatigue as a result and exercise tolerance zero.
Eventually chest pain, need to sit upright in bed (SOB).
Then a PVC/PAC and heart restores to
normalNormal saline flush function and I regain
normalNormal saline flush physical energylevels within an hour.
Episodes last for several days (when lucky only minutes) up to a week.
Sometimes, the start/stop occurs during the night: going to bed no problem, waking up with problem or vice versa.
EKGAtrioventricular block, ekg tracing
Ecg
Exercise stress test's made have only revealed IVCD and borderline abnormalities.
An echocardiogram made during an episode showed diastolic dysfunction and hyperkinesia?
Not all PVCs/PACs I have induce or terminate an episode.
No eliciting factors are known.
Holter-regs: unusual high RR-variation (way beyond normal sinus-arrhythmia from breathing), PAC's, multifocal PVC's and ns-VT's (5 seconds to 1 minute, between 160bpm-200bpm, once/twice a month).
The ns-VT's cause: near-syncope immediately following and chest pain for hours after the event.
My new EP just started another round of 'standard' investigations.
My most important questions are:
1) Could a PVC/PAC originating from a specific focus initiate and terminate some kind of alternative conduction path causing my heart to beat differently?
2) Is comparison of several echocardiograms (recorded during severe episode and when having no problems) useful?
3) Apparently symptoms are not represented on EKG. Is it possible that the above mentioned suspected conduction disturbance is hiding in a 12-lead EKG?
4) Would an experienced EP be able to find out what the mechanism is of my symptoms?
Would appreciate any information you can provide.
friendinneed