is my doctor the right one?

hi guys,,happy new year to all..just wanna have clarification about my situation before,, last may 2010 i was checked for hbv and i was clear of it..but 3 months after that was that was aug 2010 i had my medical check up and found out that i was positive for hbv.. i was hbsag reactive and hbeag reactive also. my sgpt was 86 iu/ml and hbvdna 110000000 iu/ml...my doctor told me that i was chronic hep b..and he advised me to take entacavir..now after 3 months that was nov 2010 my lab results were as follows..sgpt 48 iu/ml, hbv dna was less than 6 iu/ml..hbsag positive and hbeag negative, hbeab positive..and my doctor was quite amaze for my latest results and he could not believe it in just 3 months of medication as far as my lab results is concern..

1. am i a chronic or acute hep b as far as my medical results and my recovery is concern..
2. what is the advantage between acute and dis advantage of chronic..can both be cured?
3. what will be the best therapy in order to make hbsag negative and what will be the time format for taking all this pills cause its expensive here in the phils..

[stef2011]

and my doctor was quite amaze for my latest result

usually it takes more time but even other members of this forum had results like that on etv or tnf.one asian with the higest hbvdna reported in the billions iu/ml got response by tnf+etv.doesn t look an expert doctor or one with a lot of hbv cases

may 2010 i was checked for hbv and i was clear of it
please explain clear how hbsag negative and hbsab>10miu/ml?if not you were not clear at all

1. am i a chronic or acute hep b as far as my medical results and my recovery is concer
yo are perfectly healthy now as concern liver, just need to get alt low to less than 30 but high bmi, meat, fats, food not fresh, fatty liver, make alt higher, so it might be not the virus the reason of alt 48

as to cornic or not only hbcab igM negative can say or hbsag positive for more than 6months

2. what is the advantage between acute and dis advantage of chronic..can both be cured?
actually the advantage is not acute/cronic but pregnancy/very young infection or adult, infection as an adult is cleared 95-98% times.most of us cronic (chronic) carriers got hbv at birth we just didn t know at that time otherwise we would have been vaccinated and just discover hbv later in life

3. what will be the best therapy in order to make hbsag negative and what will be the time format for taking all this pills cause its expensive here in the phils..

well being in the philippins maybe it is not easy to find very expert and updated doctors.eradication is possible by interferon+entecavir with or without nitazoxanide, of course you need the tests to see if interferon is working, that's to say hbsag quantification according to the hbsag decrease you can say if you are responding to interferon or nitazoxanide or not.in case you have an hbsag decrease you must keep interferon as lon as hbsag gets negtive, usually 2years but in china it has been used up to 5 years

if there is no hbsag decrease just keep entecavir only until hbsag negative, a new drug or for life

all treatments for hbv are expensive everywhere in the world un fortunately and therapy can take 2years of interferon and 5-10 years of entecavir+ntz

as to drugs you must get the generics in india, as to interferon there is no way out, it is expensive, i am very sorry many country don t have healthcare and free drugs/check ups, i see india generics as the only way out

[stef2011]

2. what is the advantage between acute

of course acute clears naturally by immune system in 3-6 months as i said above in 95-98%, so no drugs/cure must be taken because they might interfere with immune systema nd there is no damge or fibrosis left after an acute hbv even if you are in the very few who have synthoms

it is not yet understood why acute with alt at 1500-3000 up to 5000 makes no damage and lower alt in cronic (chronic) does even if slowly by decades.
the latest theories are that it is not virus to make this damage but oxidative stress balance which is broken by the assault of immune system to infected cells by decades, so keeping oxidative stress to a minimum by very strong antioxidant may play a role in keeping fibrosis  to a minimum.
this theory is also aimed to explain why some develop cirrrhosis and other don t dispite high hbvdna

for example me and my sister got infected at birth probably, she has no liver damage despite hbvdna 5logs while i developped cirrhosis.the difference between us is immune activity and not virus quantity.
for example my sister has always had more virus (higher hbsag/hbvdna) than me but little immune response with no alt flares while i have always had low virus but high alt flares from time to time to keep the virus low