Aa
sex
here is a very good new paper about sex, with many citations:
http://tinyurl.com/2lf7fw
and a commentary: http://tinyurl.com/35tvwa
Have a nice weekend...
http://tinyurl.com/2lf7fw
and a commentary: http://tinyurl.com/35tvwa
Have a nice weekend...
CS, I got it from the US-commentary, see below. I agree with the comment, if you have a close look to studies, which tell us there is NO risk at all http://tinyurl.com/ysyapd you will see that in their studies were some exclusion criteria like no anal sex, no menstruation and so on. However, I think the chance to get SVR from Sex is smaller than to be hit by thunderbolt, if one followes the mentioned conditions to avoid sexual blood-blood contacts. It will be higher, if you don't follow the preconditions for unprotected sex, but the risk seems to be low, even then.
Regards, drofi
Excerpt:
The studies described above demonstrate a very low risk of sexual transmission of HCV in long-term heterosexual monogamous couples. However, these studies were conducted in predominantly low-risk populations, with reports of little (8/171 couples) [14] or no anal intercourse [12, 15] and no sexually transmitted infections (STI) or lesions [12, 15], except for HIV infection in 12 of 30 participants in 1 study [13]. Sexual transmission of HCV may occur more readily in the presence of biological cofactors, such as high-risk sexual contact or STIs that traumatize the anal or genital mucosa or behaviors that lead to blood-blood contact. Many studies of persons at risk for STIs and/or high-risk sex—such as those presenting for STI treatment [6, 19], men who have sex with men (MSM) [20, 21], and female sex workers [22, 23]—have reported independent associations of HCV with STIs and numbers of sex partners, although such associations have not been seen in all studies [24–29]. HIV status has also been frequently reported as an independent risk factor for HCV transmission after adjustment for STIs and sexual behavior [19, 30–32]. In addition, recent case studies have suggested an increased risk of sexually transmitted HCV among HIV-positive MSM in association with sexual behaviors (such as unprotected anal sex, fisting, and the use of sex toys), STIs, and noninjection drug use [33–38]. Risk-factor analyses have supported these reports [39, 40]. Several studies have also cited trends in the incidence of sexually acquired HCV infection in HIV-positive persons [41] and in HIV-positive MSM over time [42]. However, the absolute risk of infection appears to be low; the incidence of HCV in HIV-positive noninjecting MSM who reported unprotected sex in the Swiss HIV Cohort Study from 1998 to 2004 was 0.7%/year, compared with 0.2%/year in those denying unprotected sex [40], and the incidence rate in the French Prédiction du Risque Musculaire en Observationnel study of HIV-positive persons was 0.8%/year from 2003 to 2005 [41].
Regards, drofi
Excerpt:
The studies described above demonstrate a very low risk of sexual transmission of HCV in long-term heterosexual monogamous couples. However, these studies were conducted in predominantly low-risk populations, with reports of little (8/171 couples) [14] or no anal intercourse [12, 15] and no sexually transmitted infections (STI) or lesions [12, 15], except for HIV infection in 12 of 30 participants in 1 study [13]. Sexual transmission of HCV may occur more readily in the presence of biological cofactors, such as high-risk sexual contact or STIs that traumatize the anal or genital mucosa or behaviors that lead to blood-blood contact. Many studies of persons at risk for STIs and/or high-risk sex—such as those presenting for STI treatment [6, 19], men who have sex with men (MSM) [20, 21], and female sex workers [22, 23]—have reported independent associations of HCV with STIs and numbers of sex partners, although such associations have not been seen in all studies [24–29]. HIV status has also been frequently reported as an independent risk factor for HCV transmission after adjustment for STIs and sexual behavior [19, 30–32]. In addition, recent case studies have suggested an increased risk of sexually transmitted HCV among HIV-positive MSM in association with sexual behaviors (such as unprotected anal sex, fisting, and the use of sex toys), STIs, and noninjection drug use [33–38]. Risk-factor analyses have supported these reports [39, 40]. Several studies have also cited trends in the incidence of sexually acquired HCV infection in HIV-positive persons [41] and in HIV-positive MSM over time [42]. However, the absolute risk of infection appears to be low; the incidence of HCV in HIV-positive noninjecting MSM who reported unprotected sex in the Swiss HIV Cohort Study from 1998 to 2004 was 0.7%/year, compared with 0.2%/year in those denying unprotected sex [40], and the incidence rate in the French Prédiction du Risque Musculaire en Observationnel study of HIV-positive persons was 0.8%/year from 2003 to 2005 [41].
Drofi the following are the study.
Where did your conclusions come from, did I miss something. I had seen the paper before but not the commentry
Results. In multivariable analysis, significant individual risk factors for HCV included a history of injection drug use, tattooing, and older age. At the couple level, HCV infection tended to cluster within couples, and this interdependence was accounted for by couples’ drug-injection behavior.
Individual and couple-level sexual behaviour was not associated with HCV infection.
Conclusions. Our results are consistent with prior research indicating that sexual contact plays little role in HCV transmission. Rather, couples’ injection behavior appears to account for the clustering of HCV within heterosexual dyads.
CS
Where did your conclusions come from, did I miss something. I had seen the paper before but not the commentry
Results. In multivariable analysis, significant individual risk factors for HCV included a history of injection drug use, tattooing, and older age. At the couple level, HCV infection tended to cluster within couples, and this interdependence was accounted for by couples’ drug-injection behavior.
Individual and couple-level sexual behaviour was not associated with HCV infection.
Conclusions. Our results are consistent with prior research indicating that sexual contact plays little role in HCV transmission. Rather, couples’ injection behavior appears to account for the clustering of HCV within heterosexual dyads.
CS
Jeez Drofi I came away thinking dont share needles.
I thought it said there was no evidence of sexual transmission.
CS
I thought it said there was no evidence of sexual transmission.
CS
Hi,
the title was a little bit misleading, indeed :-)
Too bad, because the papers are a great overview.
Which praktical conclusion can be extracted from the paper and the commentary?
When the following conditions are given, there is no need to use condoms:
• HCV infection is chronical, infection timepoint was in the past, no acute infection
• no unprotected anal sex (threat of microlesions )
• there are no lesions of the mucosa
• no unprotected sex when menstruating
• both partners have no additonal infections or medical conditions, which could cause lesions of the mucosa (eg genital fungal infection) or an immunsuppression (eg systemic corticoid treatment).
regards, drofi
the title was a little bit misleading, indeed :-)
Too bad, because the papers are a great overview.
Which praktical conclusion can be extracted from the paper and the commentary?
When the following conditions are given, there is no need to use condoms:
• HCV infection is chronical, infection timepoint was in the past, no acute infection
• no unprotected anal sex (threat of microlesions )
• there are no lesions of the mucosa
• no unprotected sex when menstruating
• both partners have no additonal infections or medical conditions, which could cause lesions of the mucosa (eg genital fungal infection) or an immunsuppression (eg systemic corticoid treatment).
regards, drofi
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