CSF Results are in--please look! Thank you.

Here are my CSF results. Any interpretations would be greatly appreciated. Any chance this could be anything other than MS? I will be seeing the Seattle MS specialist very soon since my neuro has now referred me as a result of the csf findings. At the bottom, I have included my brain MRI results for a little background. My only major problem is severe migraines with aura and back/pinched nerve problems. I am a 33 year old female.

CSF Total Volume 4.0
Clots BE’ Clots Absent
Xanthochram CSF Absent
Tube Plum CSF 4
Color CSF Colorless
Appear CSF Clear
WEC C5F 6 H      0-5 /nim3
RBC CSF 0         0—0
Polys CSF 0      0-6
Lymph CSF 94 H 4080
Histio/Mono CSF 6 L        15—45
Irtrp BE’ See corrunent *
CSV’ Protein 16        15—50 mg/dL
CSF Glucose 62.       40—70 mg/dL
11/18/09 14:20:00 Intrp SF:
No significant inflammatory exudate and no cytologic atypia.

Multiple Sclerosis Comp Panel
Immunoglobulin Q, Serum 1230 mg/cit [768—1632]
Inimunoglobulin G, CSF 2-1 mg/cit (0.0—6.0]
Albumin, Serutu (Neph) 4460 mg/cit [3500-5200)
Albumin, CSF 8 mg/cit (0—35]
Albumin Index 1.8 ratio [0.0-9.0)
CSF Ig Synthesis Rate 1.0 mg/cit [0.0-8.0)
tgG Index H 0.95 ratio [0.28—0.663
CSF IgG/Albumin Ratio H 0.26 ratio (0.09-0.25)
CSF Oligcolonal. Bands Positive [NEG]
Myelin Basic Protein 0,45 ng/ml (0.00—1.10)

Interpretation
Increased concentrations of IgG in the CS? is an
important indicator for MS but may also be associated
with increased permeability of the blood—CS? barrier,
or increased local production cf IgG, or both.
Increased IgG production is demonstrated by an
increased CS? IgG/Albumin ratio, IgG Index and IgG
synthesis rate.
As compared to the serum, isoelectric focusing/
irnmunofixation reveals 10 IgG band(s) that is Care)
unique to the CSE This is consistent with intrathecal
synthesis of iismutoglobulin and is considered to be a
positive result for the oligoclonal bands. Oligoclonal
bands are present in over 90% of patients with MS but
may also be present in CSF from patients with viral or
bacterial meningeoencephalitis, SSPE, neutosyphilis,
Guillain—Barre syndrome, and meningeal carcinomatosis.

MRI:
In the left posterior parietal lobe, on the axial FLAIR, there is subcortical foci of white matter hypertensities. These are also left faintly visible on the axial T2 images. These areas do not definitely enhance. There is also a left splenium of the corpus callosum foci of T2/FLAIR hyperintensity on both series.
Differential would include demylinating syndrome, multiple sclerosis, vasculitis, and vasculopathy.