hi I had an mi about ten yrs ago,had angioplasty. ok for a year.developed chest pain yr ltr,went to er 4 enzyme test said another mi coming,had 3 stents(8 years ago)did fine for quite some time than was told i had gloumular nephritis..been on kidney transplant list 3 years now.last years pet scan revealed heart enlarged slightly +/murmer ive had forever since child hood/ef ratio of 54. a year later same result but ef of 45 my bp up under testing to 170/54-46 I know my heart rate is low because of labetolol 200 mg up to 6x daily (which is alot !)and slows my heart rate way down Ihave/heard/ seen 44 bpm(I can hear it when my bp is hi or low) due to aortic valve rplcment(valve is noisy) my dr says to keep bp and heart rates low to preserve valve.can it be due to labetolol or other meds?plavix 75 mg minoxidol 10 mg,lisinopril 10 mg bumex 2mg or any folic acid, b-12,renavite,asprin or my binders?phoslo,renvela?I was wondering this cause I took a labetolol just before my pet and felt awful in pet scan tube,and have crashed dyalisys mach w/low hrt beat and had to pause a while,getting a sonogram next thurs and I am worried that I will no longer be a candidate for a kidney.....thank you
What is your angio reports?
Let us know the outcome. Wish you well!
Thank you that makes me feel better, yes they did stress test which found this. All other test have been normal. Doing stress test the EF was 35% but, did echo yesterday and it was 45%. Schedule for angiogram next Tuesday but, DR doesn't think he will find blockage but, wants to be on safe side. All this came about found chest pains that would come and go.
Apparently, you have had an echo to determine your EF. Is there any hypokinesis (heart wall movement impairment), heart wall dimensions normal, heart structually normal, and any valve disorder? About 6 years ago I had an EF below 29%, enlarged heart, valve problem...today with treatment my heart size and EF is normal. But the underlying cause was ischemia (lack of blood flow) to the heart due the heavy workload of the heart pumping against occluded vessels. Medication reduced the workload and with time the heart returned to normal functionality.