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by MA PC, Sep 13, 2007 03:10PM
Two months ago (July '07) I had 2 episodes of dizziness, nausea, and vomiting (each time lasted about 3 days). On the second incident I went to my GP which led to an MRI (the imaging results are below). I then saw a neurologist who ordered a battery of labs (including Cadisil). The results showed high cholesterol and triglycerides (at which time I was put on medication for this). Today, at my follow-up appointment with the neurologist I was told that I "didn't look like I have MS"....and that it's "possible" that the high cholesterol levels could have caused my "issues." He is suggesting another MRI next month (3 months after the 1st). My question is, "if I am having no symptoms why would another MRI be indicated so soon...rather than, say 6 months?" And, if the next MRI actually showed "full blown" MS they would simply treat the symptoms, right? And since I'm having no symptoms.....
During this process it was found that I have some endocrine issues (multinodular goiter -- am scheduled for some blood tests in the next few days) and degenerative cervical spine.
Thank you so much for your time.
MRI Findings
History: 54-year-old female with dizziness, headache, and visual
changes, as well as nausea and vomiting.
Technique: The following imaging sequences of the brain were
obtained: Sagittal T1, axial diffusion weighted imaging, axial
FLAIR, axial T2, axial T1, and sagittal FLAIR. Following
injection of 20 cc gadolinium IV contrast, axial T1 imaging was
performed.
FINDINGS:
Cerebral volume is age-appropriate. No intracranial hemorrhage or
extra-axial fluid collection is demonstrated. The ventricles and
cisterns are patent, without hydrocephalus or herniation.
Moderate supratentorial white matter disease, manifested by
numerous small foci of T2 and FLAIR hyperintensity, reveal no
abnormal enhancement or restricted diffusion of acute ischemia or
acute inflammation. While the differential diagnosis for white
matter signal changes include several entities, most commonly
chronic microvascular ischemic change, with additional entities
such as cerebral vasculitis, sequela of migraine headaches, and
infectious etiologies, the sagittal FLAIR images suggest a
subcallosal striation pattern and Dawson's fingers pattern which
is fairly characteristic of multiple sclerosis, though this
entity is a clinical diagnosis. No infratentorial white matter
disease is delineated.
No focal pituitary gland abnormality or cerebellar tonsillar
herniation. Grossly normal internal auditory canals. No apparent
vascular occlusion.
Grossly well aerated paranasal sinuses and mastoid air cells. No
significant abnormality of the orbits or imaged osseous
structures.
IMPRESSION:
Moderate supratentorial white matter disease in a pattern fairly
suggestive of multiple sclerosis, for which correlation with
clinical factors is recommended. No associated enhancement or
restricted diffusion of acute inflammation.
During this process it was found that I have some endocrine issues (multinodular goiter -- am scheduled for some blood tests in the next few days) and degenerative cervical spine.
Thank you so much for your time.
MRI Findings
History: 54-year-old female with dizziness, headache, and visual
changes, as well as nausea and vomiting.
Technique: The following imaging sequences of the brain were
obtained: Sagittal T1, axial diffusion weighted imaging, axial
FLAIR, axial T2, axial T1, and sagittal FLAIR. Following
injection of 20 cc gadolinium IV contrast, axial T1 imaging was
performed.
FINDINGS:
Cerebral volume is age-appropriate. No intracranial hemorrhage or
extra-axial fluid collection is demonstrated. The ventricles and
cisterns are patent, without hydrocephalus or herniation.
Moderate supratentorial white matter disease, manifested by
numerous small foci of T2 and FLAIR hyperintensity, reveal no
abnormal enhancement or restricted diffusion of acute ischemia or
acute inflammation. While the differential diagnosis for white
matter signal changes include several entities, most commonly
chronic microvascular ischemic change, with additional entities
such as cerebral vasculitis, sequela of migraine headaches, and
infectious etiologies, the sagittal FLAIR images suggest a
subcallosal striation pattern and Dawson's fingers pattern which
is fairly characteristic of multiple sclerosis, though this
entity is a clinical diagnosis. No infratentorial white matter
disease is delineated.
No focal pituitary gland abnormality or cerebellar tonsillar
herniation. Grossly normal internal auditory canals. No apparent
vascular occlusion.
Grossly well aerated paranasal sinuses and mastoid air cells. No
significant abnormality of the orbits or imaged osseous
structures.
IMPRESSION:
Moderate supratentorial white matter disease in a pattern fairly
suggestive of multiple sclerosis, for which correlation with
clinical factors is recommended. No associated enhancement or
restricted diffusion of acute inflammation.
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