May 12, 2009 10:04PM
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I'll be starting Chlorozoxazone today. My neurologist who is a movement disorders specialist agreed to start me on it. Thank you for providing me with the information. I will be taking it under his guidance and transferring from the Flexaril to it over the coming days. I had a blood test for liver function at the office to check up on that. The movement disorders specialist informed me that Piracetam (the natural remedy) works like the anti-convsulsant Keppra and is an an anti-seizure drug in Europe. Keppra is used for tardive dyskinesia as well so that makes sense. Now as for the "unknowns" well he suggested Namenda for tardive dyskinesia. It also works on cogntion (so it would improve the tardive dysmentia) and is an experimental mood stabilizer. But Namenda modulates NMDA receptors in the exact opposite manner as glycine so that's two "unknowns". Well I asked my psychopharmocologist to be in touch with the glycine researchers and asked if the two have ever been given together. I await an answer and there might not be one as of yet. We'll see.
This is the interesting part. I had as stated documented the tardive dysphrenia, tardive psychosis and tardive dysmentia on a day when they were at their worst. I was experiencing full dissociation. It wasn't "the match girl". Its obviously a full neurological disruption as there were lights and flames that I saw. I didn't "believe" them. They were seizure like but epilepsy (and all common criteria) had been ruled out. Dysphoric mania. And various tic like compulsions (tardive tourretecism). I had no idea how he would take my briefing for the case study I wrote that day. He accepted it. And the visit ended with a hand shake. That's an agreement. Just like my psychopharmocologist I'll be working with him on a higher level. Now the thing to do is give them some breathing room and let them discuss the case study and revise it within standard clinical terminology. As to what happens from there much is confidential. What I can say is the PET scan is starting to become more of a potential. Its an issue of coverage and who will field it. The ideas that I worked to identify are gradually being incorporated into clinical understanding. And will benefit others. I may not be aware of all of their discussions. And not every study will be a success. But the idea that tardive pyschosis, tardive dysphrenia and tardive dysmentia could be spoken about in a "non controversial" manner is appearing to be realistic. Identify, prevent and treat them. And they are treatable And as for the glutamate antagonists as antipsychotics much is being said about that. And the idea that people could and should have the mental recovery I did without the neurological disabilities is something that many people would like to see. And for the researchers who achieve it as well as the ones who worked on it I thank you for your help in everyone's recovery and am grateful I could be a part of this.