Can you describe the biochemistry/pathophysiology of hematoma resolution.

[dpdoc]

Not much information in the literature on this subject as this is extravascular and he traditional discussions regarding clot resolution are inadequate at best.  I am looking for the chain of events that ultimately results in the disposal of the extravascular iron deposits resulting from a simple bruise of the upper extremity. Hemosiderin, a globular protein,  the primary Fe store must be denatured in some way to release the stored iron.  Just unclear as to how the iron is released and how it is cleared from the extravascular space.  Some say it is transported to the kidney for excretion, but by what mechanism is the iron transported, as it resides outside the vasculature in the extravascular space.  
Just thinking a Dermatologist might shed some light on the subject.  This is not a straight forward question and if too much or inappropriate for this venue, I understand, and thanks for your consideration