Aa
Ear and Jaw Pain
For approx 5 months I have had severe internal ear pain, which has now moved down into my jaw - I have been on (2) antibiotics, sudafed and claritin with no relief - I have been to an ENT and family doc and they say that it is allergies....I do not think so. I am at a loss and do not know where to go from here. If anyone has advice, I'd truly appreciate it. My fear is the longer the pain continues and grows, the worse the condition will get. I look forward to a response. Thank you and God Bless!
I have exactly the same problem. It started about 8month ago. Now the pain inside ear is stronger and pain ander the jaw. But, it is not constant. I have a lot of stress at work now and think that it can be the stress. I went to 2 ENT, did MRI, wnet 5 times to my enternal and no one found anythihg. My entrnal prescribed me Mobic antiinflamattory drug. I will try it.Also, the area around the ear feels tender. It all drives me crazy since no one has any answer.
Hi, have you looked into this?
EAGLE'S SYNDROME - SYMPTOMS
Patients with vague head and neck pain symptoms can lead to an extensive differential diagnosis. One easily overlooked but important cause of chronic pain is Eagle’s syndrome (ES). Beginning in 1937, Dr. Watt Eagle published a series of articles describing a constellation of symptoms associated with an elongated styloid process. This syndrome that bears his name is associated with two classic presentations. The first, which the otolaryngologist is more familiar with, is throat pain radiating to the ear in a post-tonsillectomy patient. The second, and lesser-known presentation, is constant throbbing pain throughout either the internal or external carotid artery distributions.
A patient exhibiting the symptoms associated with Eagle’s syndrome, may consult their family physician or an otolaryngologist, a neurologist, a surgeon (neurosurgeon, maxillofacial or oral surgeon), a dentist or even a psychiatrist in order to be diagnosed. Persistent pain and other symptoms could aggravate the psychological state of a patient. By the time the syndrome is actually diagnosed, many patients have already visited some of these doctors, who have unsuccessfully tried to treat their symptoms.
Symptoms depend on a variety of factors, including the length and width of the styloid process, the angle and direction of its deviation and the degree of ossification. The pathogenesis of the syndrome was described by Eagle, who discussed types.
The first type, ‘‘classic Eagle’s syndrome,’’ typically occurs in patients after tonsillectomy, although it can also occur after any other type of pharyngeal surgery. A palatable mass may be observed in the tonsillar fossa, its palpation sometimes exacerbating the patient’s symptoms. Symptoms include ear pain, neck pain extending to the oral cavity and the maxilla, dysphonia, dysphagia, odynophagia, persistent sore throat, the sensation of a foreign body in the pharynx, painful trismus <25 mm, vertigo and tinnitus. Pain is also observed when turning the head or extending the tongue. Apart from turning the head, yawning can also trigger symptoms, particularly those resembling migraine. Other symptoms may include tongue pain in general, a sensation of increased salivation, alterations in taste, vocal changes, pain in the upper limbs, chest, and temporomandibular joint, facial paresthesia, pharyngeal spasm, pain triggered by the movement of the mandible, cough, dizziness, or sinusitis. Eagle’s syndrome has also been reported as the most important cause of secondary glossopharyngeal neuralgia or atypical craniocervical pain. All of these symptoms are attributed to the irritation of cranial nerves V, VII, IX or X, all of which are situated very close to the styloid process. The observation of symptoms after tonsillectomy generates the hypothesis that these nerves are entrapped in the locally formed granular tissue. Trauma to the soft tissues during tonsillectomy may cause bone formation, leading to an elongated styloid process or ossified stylohyoid ligament. Ossification typically appears from 2 to 12 months after the trauma.
In the stylo-carotid artery syndrome, an elongated styloid process deviating slightly from its normal direction can impinge the internal or external carotid artery, stimulating the sympathetic nerve plexus accompanying the artery and causing pain during artery’s palpation. Stimulation of the internal carotid artery causes pain along the artery that is sometimes accompanied by pain in the eye and parietal cephalalgia. These symptoms can result in wrong diagnoses, such as cluster headache or migraine. Symptoms may also include aphasia, sight disturbances, weakness or even syncope episodes. Stimulation of the external carotid artery causes facial pain, mainly in the area under the eyes. Histological examination of the vessel wall in such cases may reveal arteriosclerosis. Stylo-carotid artery syndrome might also result in arterial variation.
DIAGNOSIS
The diagnosis of Eagle’s syndrome is based on four different parameters:
(1) clinical manifestations
(2) digital palpation of the process in the tonsillar fossa
(3) radiological findings and
(4) lidocaine infiltration test. During the lidocaine infiltration test, lidocaine anesthetic is administered to the area where the styloid process is palpable in the tonsillar fossa. If the patient’s symptoms and local sensitivity subside the test result is considered positive and Eagle’s syndrome is diagnosed.
A patient exhibiting the symptoms associated with Eagle’s syndrome, may consult their family physician or an otolaryngologist, a neurologist, a surgeon (neurosurgeon, maxillofacial or oral surgeon), a dentist or even a psychiatrist in order to be diagnosed. Persistent pain and other symptoms could aggravate the psychological state of a patient. By the time the syndrome is actually diagnosed, many patients have already visited some of these doctors, who have unsuccessfully tried to treat their symptoms. This is quite understandable considering that the clinical manifestations of Eagle’s syndrome resemble those of many other diseases. Consequently, it is quite difficult to make a correct diagnosis based solely on clinical manifestations. However, it is very important for physicians and dentists to include Eagle’s syndrome in their differential diagnosis when treating patients experiencing pain in the cervicofacial and cervicopharyngeal regions.
A 3D-CT scan is considered the gold standard in the radiological diagnosis of Eagle's Syndrome. It provides an accurate measurement of the length and angulation of the styloid process and is considered to be the best supplement to the plain x-ray.
It is important to note that an elongated styloid process does not necessarily signify Eagle’s syndrome, as the majority of individuals exhibiting this anatomical anomaly experience no symptoms. Additionally, although an elongated process is found bilaterally in most cases, patients typically display unilateral symptoms. Also it is noteworthy that the occurrence of the syndrome correlates with the length of the styloid process, its width and its angulation. In fact a number of mechanisms can result in the onset of the syndrome and are responsible for the variety of symptoms. Consequently, patients may experience any number of symptoms, which often mislead physicians and necessitate the use of other data such as radiological findings to confirm the diagnosis. Both physicians (head and neck, oral and maxillofacial surgeons) and dentists must have a high index of suspicion for this clinical entity. Eagle’s syndrome should be included in the differential diagnosis of cervicofacial and pharyngeal pain. The fact that it is often excluded in such cases results in underdiagnosis and, consequently, an underestimation of the incidence of this syndrome.
EAGLE'S SYNDROME - SYMPTOMS
Patients with vague head and neck pain symptoms can lead to an extensive differential diagnosis. One easily overlooked but important cause of chronic pain is Eagle’s syndrome (ES). Beginning in 1937, Dr. Watt Eagle published a series of articles describing a constellation of symptoms associated with an elongated styloid process. This syndrome that bears his name is associated with two classic presentations. The first, which the otolaryngologist is more familiar with, is throat pain radiating to the ear in a post-tonsillectomy patient. The second, and lesser-known presentation, is constant throbbing pain throughout either the internal or external carotid artery distributions.
A patient exhibiting the symptoms associated with Eagle’s syndrome, may consult their family physician or an otolaryngologist, a neurologist, a surgeon (neurosurgeon, maxillofacial or oral surgeon), a dentist or even a psychiatrist in order to be diagnosed. Persistent pain and other symptoms could aggravate the psychological state of a patient. By the time the syndrome is actually diagnosed, many patients have already visited some of these doctors, who have unsuccessfully tried to treat their symptoms.
Symptoms depend on a variety of factors, including the length and width of the styloid process, the angle and direction of its deviation and the degree of ossification. The pathogenesis of the syndrome was described by Eagle, who discussed types.
The first type, ‘‘classic Eagle’s syndrome,’’ typically occurs in patients after tonsillectomy, although it can also occur after any other type of pharyngeal surgery. A palatable mass may be observed in the tonsillar fossa, its palpation sometimes exacerbating the patient’s symptoms. Symptoms include ear pain, neck pain extending to the oral cavity and the maxilla, dysphonia, dysphagia, odynophagia, persistent sore throat, the sensation of a foreign body in the pharynx, painful trismus <25 mm, vertigo and tinnitus. Pain is also observed when turning the head or extending the tongue. Apart from turning the head, yawning can also trigger symptoms, particularly those resembling migraine. Other symptoms may include tongue pain in general, a sensation of increased salivation, alterations in taste, vocal changes, pain in the upper limbs, chest, and temporomandibular joint, facial paresthesia, pharyngeal spasm, pain triggered by the movement of the mandible, cough, dizziness, or sinusitis. Eagle’s syndrome has also been reported as the most important cause of secondary glossopharyngeal neuralgia or atypical craniocervical pain. All of these symptoms are attributed to the irritation of cranial nerves V, VII, IX or X, all of which are situated very close to the styloid process. The observation of symptoms after tonsillectomy generates the hypothesis that these nerves are entrapped in the locally formed granular tissue. Trauma to the soft tissues during tonsillectomy may cause bone formation, leading to an elongated styloid process or ossified stylohyoid ligament. Ossification typically appears from 2 to 12 months after the trauma.
In the stylo-carotid artery syndrome, an elongated styloid process deviating slightly from its normal direction can impinge the internal or external carotid artery, stimulating the sympathetic nerve plexus accompanying the artery and causing pain during artery’s palpation. Stimulation of the internal carotid artery causes pain along the artery that is sometimes accompanied by pain in the eye and parietal cephalalgia. These symptoms can result in wrong diagnoses, such as cluster headache or migraine. Symptoms may also include aphasia, sight disturbances, weakness or even syncope episodes. Stimulation of the external carotid artery causes facial pain, mainly in the area under the eyes. Histological examination of the vessel wall in such cases may reveal arteriosclerosis. Stylo-carotid artery syndrome might also result in arterial variation.
DIAGNOSIS
The diagnosis of Eagle’s syndrome is based on four different parameters:
(1) clinical manifestations
(2) digital palpation of the process in the tonsillar fossa
(3) radiological findings and
(4) lidocaine infiltration test. During the lidocaine infiltration test, lidocaine anesthetic is administered to the area where the styloid process is palpable in the tonsillar fossa. If the patient’s symptoms and local sensitivity subside the test result is considered positive and Eagle’s syndrome is diagnosed.
A patient exhibiting the symptoms associated with Eagle’s syndrome, may consult their family physician or an otolaryngologist, a neurologist, a surgeon (neurosurgeon, maxillofacial or oral surgeon), a dentist or even a psychiatrist in order to be diagnosed. Persistent pain and other symptoms could aggravate the psychological state of a patient. By the time the syndrome is actually diagnosed, many patients have already visited some of these doctors, who have unsuccessfully tried to treat their symptoms. This is quite understandable considering that the clinical manifestations of Eagle’s syndrome resemble those of many other diseases. Consequently, it is quite difficult to make a correct diagnosis based solely on clinical manifestations. However, it is very important for physicians and dentists to include Eagle’s syndrome in their differential diagnosis when treating patients experiencing pain in the cervicofacial and cervicopharyngeal regions.
A 3D-CT scan is considered the gold standard in the radiological diagnosis of Eagle's Syndrome. It provides an accurate measurement of the length and angulation of the styloid process and is considered to be the best supplement to the plain x-ray.
It is important to note that an elongated styloid process does not necessarily signify Eagle’s syndrome, as the majority of individuals exhibiting this anatomical anomaly experience no symptoms. Additionally, although an elongated process is found bilaterally in most cases, patients typically display unilateral symptoms. Also it is noteworthy that the occurrence of the syndrome correlates with the length of the styloid process, its width and its angulation. In fact a number of mechanisms can result in the onset of the syndrome and are responsible for the variety of symptoms. Consequently, patients may experience any number of symptoms, which often mislead physicians and necessitate the use of other data such as radiological findings to confirm the diagnosis. Both physicians (head and neck, oral and maxillofacial surgeons) and dentists must have a high index of suspicion for this clinical entity. Eagle’s syndrome should be included in the differential diagnosis of cervicofacial and pharyngeal pain. The fact that it is often excluded in such cases results in underdiagnosis and, consequently, an underestimation of the incidence of this syndrome.
I do have the same type of problem, Inner Ear pressure, Neck and Headaches, A sore thraot and only now the jaw pain/pressure is extra, but it is a definate allergyto me, physio don't help stress relievers a bit, but an Anthistamien is the answer, takes a while before it clears up and then stay away for a while till the next one hits you .....
hey rae, are you stressed? do you clench your jaw? is your jaw feeling very tender around your ears? it seems you have a bit of what i think i have which is anxiety induced jaw / ear pain. i'm trying to massage my jaw lightly as i think one of my biggest issues is sleeping, and clenching my jaw while I sleep (which also affects my ears). i'm also going to the chiropractor to help with my neck... and about to schedule a dentist appointment so they can maybe take an xray, and see whther my jaw is misaligned. anyway, some areas for you to consider and check ;-)
good luck, and don't give up!
good luck, and don't give up!
Have an Answer?
You are reading content posted in the Ear, Nose & Throat Community
Think a loved one may be experiencing hearing loss? Here are five warning signs to watch for.
Discover the common causes of and treatments for a sore throat.
Learn about what actually causes your temperature to spike.
Find out which foods you should watch out for.
Family medicine doctor Enoch Choi, MD helps differentiate between the common cold and more threatening (bacterial) infections
Dr. Steven Park reveals 5 reasons why breathing through your nose could change your life
