Inverted (or negative) T waves can be a sign of coronary ischemia. - 0.1 micro volt T wave is inverted for leads V3-V6, etc.
PR interval 198 ms is normal.
QRS duration is 102ms...normal is 60-100ms.
The duration, amplitude, and morphology of the QRS complex is useful in diagnosing cardiac arrhythmias, conduction abnormalities, ventricular hypertrophy, myocardial infarction, electrolyte derangements, and other disease states.
Long QT can be acquired and more common than the various congenital causes of long QT duration described above, are acquired causes. They can be divided into two main categories - those due to disturbances in blood electrolytes and those due to various drugs.
Inverted T waves indicates some vessel blockage in the locations described. It is possible there is restenosis with the stent or occlusion in the other area described may be causing angina (chest pain) due ischemia (lack of blood flow) to the area described.
msThankyou for your reply so quickly and has clariffied that matter. However after reading my patient cardio card I have come to see other areas that I'm not sure what they mean.
Twave abnormality possible anterolateral ischema -0.1+ MV T wave in V3-V6
T wave abnormality, possible inferior Ischemia -0+MV T wave in 11/AVF
Vent rate: BPM
PR int: 198 ms
QRS dur: 102ms
P-R-T axes: 43 10 172
QTcB: 571 ms
Down the bottom of ecg it has:
O version 1.04.17 Sequence 06425 25mm/s 10mm/mV 0.05-150 Hz
Enzymes were 0.3
I've had a stent put in front artery which they said was narrowed but not blocked.
On discharge have had slight burning in chest occasionally but eases from day, could this be due to stent?
In the strict sense of the words "heart attack" medically termed an infarct means necrosis in the tissue due to local ischemia (means obstructed blood flow and a heart attack is occurring with necrosis of heart cells...this condition can be quick from a clot or a slower process with occlusions of coronary vessels.
An EKG is not a very reliable source to detect heart muscle damage and a prior heart attack. However,non-ST segment elevation myocardial infarction (NSTEMI) is a heart attack, or MI (myocardial iinfarction), and there should have been chemical markers in the blood that indicate damage has occurred to the heart muscle. In NSTEMI, the blockage may be partial or temporary, and so the extent of the damage is relatively minimal.