plaque reversal

[nahm]

one yaer back i underwent angioplasty with one medicated stent inserted in lad which was 90% blocked.
AGE 58 MALE., NO BP, NON DIABETIC, WT 150LBS, HT 5' 9", ONE KIDNEY FUNCTIONING.
PRESENTLY ON PLAVIX 75, LIPIGET 40, ASPRIN 75.
TOTAL CHOLESTROL 110, LDL 40, HDL 58, TRIGL 90

WITH ABOVE CONDITION ANY CHANCE OF PLAQUE REVERSAL IN OTHER ARTERIES WHICH ARE 30% BLOCKED. IF YES HOW MUCH TIME IT CAN TAKE.

Thanks NAH

[ed34]

Yes I did see some about vitamin K but they were years ago and I was waiting to see if this new 'knowledge' would hit the medical community. Nothing so far, not even a hint. So, I am wondering if the results are as really as stated, but who knows.All my blood tests haven't revealed any issues yet regarding vitamin K and so the question is, are all inhabitants of developed countries deficient which we have regarded as normal levels? or does pumping extra K into the body have other side effects yet to be realised?

[Hofbrain]

Hi Ed,

Have you come across any studies of Vitamin K2?  I have seen a couple whereby calcium is removed from the bloodstream and deposited into the bones.

Steve

[erijon]

What Ed says is correct. I have read studies that state having an LDL under 70 along with statin use and regular, daily aerobic exercise can cause regression in blockages. Here is the link;

http://www.ncbi.nlm.nih.gov/pubmed/20234098

This is from the National Institute of Health (NIH). I work with many cardiologists as a volunteer and all have told me they have never seen evidence of regression. The studies have been limited in size so they may not be dependable, hard to say.

Jon

[ed34]

While there are a few reports floating around the internet that claim regression of plaque has been observed, every cardiologist I've seen has never observed this. They have seen the odd few who have stopped progression. From what I understand, there is nothing in the blood to mop up pure fat. Fat cannot be dissolved in water and blood is mostly water, and this is why fat starts off inside lipids to transport it, and has to be moved around the body in lipids. I don't think a lipid has the ability to go up to pure fat and absorb it. When white cells enter the damaged area of artery, they are injected with fat and they become large macrophages. Damaged by free radicals, the macrophages can't signal to have their fat removed again. So they die and turn into foam cells, basically a soft fat. They are now out of the natural process. That;s how I understand it.
However, if the blockage is mostly calcium then perhaps there is a mechanism the body has for reabsorbtion of this? The calcium can be acting as a protecting barrier, for soft fat underneath, so removing the calcium COULD let this escape and cause blockages. The whole thing is very complex and still not fully understood. Only fairly recently have they discovered the immune system is partly controlled by HDL lipids. So, perhaps these aren't the good guys after all. They control the macrophage conversions and call white cells to the area in the first place.