As far as i'm aware MS has no connection at all with true Parkinson's, having a family history of Parkinsons would mean there 'could be' a higher probability of Parkinsons because of the known genetic associations but i can't come up with any reason why there would be 'any' probability of other family members having MS instead....
Keep in mind that Parkinson's is caused by the decline in the brain's production of the chemical dopamine, the Basal Ganglia located in the middle of the brain is where many of the cells are which produces dopamine. Parkinsons is a very slow development of very telling symptoms and it typically takes a lot of years before these issues become disabling, the average age of dx is actually over 65, early onset Parkinson's is actually uncommon and usually much slower in younger people.
There are still no specific tests to confirm Parkinson's, dx is clinical so based on the combination of symptoms and their behavior patters eg at rest tremor, Bradykinesia (slowness), Dystonia, pill rolling tremor, soft speech, freezing, limb rigidity etc and also ruling out all the alternative causes including medications, psychological, other movement disorders etc etc.
Over the years we've probably had some people dx with Parkinson's or Parkinsonism because of another medical conditions but i can't think of anyone still here or anyone dx'ed with MS who's actually mentioned having a family history of Parkinson's sorry...
What's got you thinking about Parkinson's?
Hope that helps....JJ
Thank you for your comment.Iam thinking about that because my
Thank you friend for your comment.Iam thinking about that because my mother suffers from Parkinson's and has become in the last stage of the disease, although the symptoms began only seven years ago.
Your professional responses always helpful to me and I appreciate it. Yes, unfortunately my mom is not a good candidate for DBS.
To return to the MS, I have many symptoms that are similar to MS, so I'm afraid.
The MRI report without contrast says:
Findings: There are small T2W/FLAIR hyper intensities foci changes in both hemispheres located mainly in the white matter in juxtacortical region. The largest one measures 4 mm and seen in the left parietal lob No one of these lesions shows restriction on diffusion. These finding are most likely compatible with chronic ischemic changes.
Conclusion: Some hyperintensities T2W/FLAIR changes in both hemispheres. These Lesions have aspecific significant (gliosis) but follow up and neurologist consultation are advised to exclude demyelinating disease. Right maxillary sinus retention cyst.
No cervical Lesions.
EMG is normal.
The neurologist said that there is no MS, and my symptoms are because of stress.
Iam afraid because according to the McDonald criteria, juxtacortical lesions are specific to MS. And I can’t believe that stress can cause all my symptoms.
I got another consultation and it was similar with the first one.
What makes me more afraid that many of the patients in this forum have been diagnosed with the disease after several years although the first diagnosis was not Ms.
What do you think??
Thank you for your help
Thank you very much for the explanation.
But when these lesions are present, and the symptoms are very similar to MS, so why he didn't consider that as a suspected MS.
I have also a Neurogenic bladder and the doctor did not care of it.
You've got more than enough reason to be 'stressed' and mental health can mimic neurological 'symptoms', so M/H 'could be' a legitimate medical causation but keep in mind that whilst symptoms can seem the same or similar, mental health and neurologically caused symptoms have specific characteristic features on clinical examination and understanding why both neurologist believe your symptoms are mental health would probably be very helpful to you....do you know what, if any abnormal clinical signs were found during your neurological assessment?
Information: Juxtacortical brain lesions
"The term juxtacortical is most commonly encountered in the brain in the context of multiple sclerosis, and is often poorly understood and misused. It is particularly important in the context of McDonald criteria, although oddly enough the term is not defined in either the original 2001 paper or the 2010 revision 1,2. Perhaps partly due to this, it is a term often misunderstood and thought to mean merely "sort of near the cortex". This is not the case, but rather it refers to lesions which abut the cortex 3, and thus involve the subcortical u-fibres. Another term which is perhaps more easily understood is leukocortical (i.e. involving both cortex and juxtacortical white matter). "
I agree with what imm's pointed out and just wanted to add, that when ever a mental health causation has been placed on someones potential causation list, I always advice they get their mental health appropriately assessed by a psychiatrist or psychologist, as it's always in your best interest to get it confirmed or proven to not be mental health related, either outcome could be very helpful in the long run....
Hope that helps.......JJ
Thank you my friends for helping and clarifying some issues.
I will go to a brain & cervical MRI next month to make sure that there is no dissemination in time.
last question. Some studies say that high stress and tension for a long period may cause MS, is this true??