Jan 10, 2012
History: 41-year-old woman with eye and facial pain, right foot
cramping and weakness. Double vision, tremors. Decreased left lower
Comparison: MRI brain 6/30/2011.
Technique: Multiplanar multisequence MRI of the brain were obtained
before and after the uneventful administration of 13 cc IV Magnevist.
DTI sequences were submitted, axial D WI, axial T1, axial T2 and
FLAIR, T2 spin-echo, sagittal T2 FLAIR and postcontrast T1-weighted
Findings: Diffusion-weighted sequence demonstrate no evidence for
Multiple T2 hyperintense abnormalities are demonstrated within the
supratentorial white matter with relative sparing of the periependymal
margin and corpus callosal white matter. 9 mm focus of T2
hyperintensity demonstrated in the deep white matter of the
MID/POSTERIOR left temporal lobe. There is a tiny focus of T2
hyperintensity within the undersurface of the left corpus callosal
body (sagittal image 19 series 13). There is a tiny focus of T2
hyperintensity within the right superior frontal gyrus (axial image 22
series 10). Focus of T2 hyperintensity demonstrated in the subcortical
white matter of the left middle frontal gyrus (axial image 17 series
10). Largest T2 hyperintense lesion in the LEFT TEMPORAL LOBE is
stable compared to outside prior MRI 6/30/2011. Small foci of T2
hyperintense described above are not demonstrated on prior
examination, likely due to significant differences in acquisition
technique and magnetic field strength.
Brain stem, and cerebellum are unremarkable. Size and configuration
of the ventricles are normal.
T1 spin-echo images shows no areas of abnormalities. Postcontrast
sequences shows no evidence for abnormal enhancement.
Orbits, optic nerve sheath complex, optic chiasm and pituitary fossa
are unremarkable. Mucous retention cyst in the right maxillary sinus.
IMPRESSION: Accounting for differences in acquisition technique and magnetic field
strength. No convincing evidence for significant interval change when
compared to 6/30/2011. Scattered T2 hyperintense lesions in the
cerebral white matter which are nonspecific for multiple sclerosis. No
abnormal enhancement to suggest active demyelination.