I had PRK in 1996 for -10 OS and -12 OD. Had cataract surgery OD June 2014 and a ZCBOO +27 monofocal Technis lens was implanted. Yag August 2014. Prior to cataract surgery, uncorrected 20/40 vision. Post cataract surgery 20/200. Immediately after cataract surgery noticed loss of detail and contrast, poor visual quality with low light including indoor light. Difficult night driving. Drops to constrict pupil did not help.
Can not see facial details inside areas even the size of my living room even with glasses. Dr. who did the cataract surgery stated that is because my "cornea is ruined" from the amount of PRK in 1996. I had retinal consult to R/O
any problems in that respect - he found things to be ok as far as he was concerned. Had much trouble with depth perception. Tried RGP lens but too uncomfortable and did not help lowlight vision. Had 2nd PRK in April 2015 with a different MD. Central cornea 413 pre-PRK and 383 post-PRK and about 20/30. Helped with depth perception, but not with details, contrast, and low light vision. March 2016 PRK regressed to 20/50. Prednisone taper got it back to 20/30 but IOL clouded again and had 2nd YAG April 2016. December 2016 vision is 20/40 - 20/50. Corneal specialist feels it could be do to high amount of corneal aberrations - feels topo-guided PRK may help visual quality. Also feels steep ablation zone maybe why it keeps regressing. I did see a 2nd retinal specialist and he felt the diameter of the IOL in relation to my high myopia maybe a factor - that some light is not refracted back to my retina as well as a larger diameter lens could do and that possibly an IOL exchange with larger diameter lens could help. Glaucoma specialist does not feel its a glaucomatous field but possibly one time ON assault due to pressure during cataract surgery. She did note inf TI defects OD. My eye has had a sore, irritated feeling since the cataract surgery and red.
My questions are: ---By not doing IOL exchange that the retinal MD referred to, could the lack of light to the retina result in my brain shutting down vision to that eye or impair the the retina in some way neurologically speaking?
-Has anyone had experience with an IOL clouding twice?
-Could the IOL be reversed and is there any definitive way to tell? I have been told that is not possible for it to be reversed and even if it were it would not matter with this type of lens. I did read on one site that this could impair scoptic vision even with this type of lens.
-I did have a UBM and was assured IOL is in place but am wondering
what could account for the inf TI defects and could it be determined from UBM if IOL is reversed?
-What are your opinions on topo-guided PRK in my case?
Thanks for any help