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Oral and kissed
The conclusion that Gillisson do is a little strange, for those who had sex've probably also less likely kissed a lot of people, and those who have had sex probably has considerable experience of kissing, a Finnish study also suggests that oral HPV at one spouse increases the risk for oral HPV in other spouse. Then she misses age oral HPV is common in the elderly, those who have not had sex are usually younger.
She also writes in later studies that no specific sexual activity can be directly linked to oral HPV, they do not really know what o how it infects, recent surveys that you can google up shows a very strong correlation between periodontal disease and oral HPV, something that makes it can infect and gain access to the basal layer, but not least infection that makes the infection becomes persistent, periodontal disease is very rare in young people, people who do not smoke or drink, and it is in this group that while kissing is rarer that when conclude that it is the absence of kissing that makes you not have oral HPV, when so many other things come into play ......... Only HPV is not likely to cause cancer, even if the connection is hpv cancer does not mean that the link-hpv kissing or oral sex, is strong
She also writes in later studies that no specific sexual activity can be directly linked to oral HPV, they do not really know what o how it infects, recent surveys that you can google up shows a very strong correlation between periodontal disease and oral HPV, something that makes it can infect and gain access to the basal layer, but not least infection that makes the infection becomes persistent, periodontal disease is very rare in young people, people who do not smoke or drink, and it is in this group that while kissing is rarer that when conclude that it is the absence of kissing that makes you not have oral HPV, when so many other things come into play ......... Only HPV is not likely to cause cancer, even if the connection is hpv cancer does not mean that the link-hpv kissing or oral sex, is strong
Abstract
BACKGROUND:
The natural history of genital human papillomavirus infection is well known, but nearly nothing is known about the outcome of oral HPV-infection.
OBJECTIVES AND STUDY DESIGN:
To study natural history of oral HPV in spouses during the follow-up 331 women (mean 25.5+/-3.4 years) and 131 men (mean 28.8+/-5.0 years) were recruited from maternity unit. Scrapings from healthy oral mucosa of spouses at baseline, 2, 6, 12 and 24 months and genital samples were taken for HPV testing. HPV DNA was detected by nested PCR and confirmed by hybridization using a cocktail of 12 high-risk (HR) oligoprobes.
RESULTS:
The detection rate of HR HPVs varied from 15% to 27%. Baseline oral HPV status between the spouses was closely related (odds ratio 4.3; 95% confidence interval 1.6-12.0; P=0.006). Persistent oral infection in one spouse was a significant risk factor (odds ratio 10.0; 95% confidence interval 1.5-68.7; P=0.005) for oral HR HPV persistence in the other partner. Cumulative incidence of new HR HPV infections was identical in both spouses, while men seemed to clear their infection more rapidly. In univariate survival analysis, the partner's oral or genital HPV status, oral sex habits or age did not predict clearance or acquisition of oral HR HPV.
CONCLUSION:
Natural history of HPV infection in oral mucosa mimics that of genital HPV infection. Oral sex had no association to oral HPV infection, but a persistent oral HPV infection of the spouse increased the risk of persistent oral HPV infection 10-fold in the other spouse.
PMID:
16112613
[PubMed - indexed for MEDLINE]
BACKGROUND:
The natural history of genital human papillomavirus infection is well known, but nearly nothing is known about the outcome of oral HPV-infection.
OBJECTIVES AND STUDY DESIGN:
To study natural history of oral HPV in spouses during the follow-up 331 women (mean 25.5+/-3.4 years) and 131 men (mean 28.8+/-5.0 years) were recruited from maternity unit. Scrapings from healthy oral mucosa of spouses at baseline, 2, 6, 12 and 24 months and genital samples were taken for HPV testing. HPV DNA was detected by nested PCR and confirmed by hybridization using a cocktail of 12 high-risk (HR) oligoprobes.
RESULTS:
The detection rate of HR HPVs varied from 15% to 27%. Baseline oral HPV status between the spouses was closely related (odds ratio 4.3; 95% confidence interval 1.6-12.0; P=0.006). Persistent oral infection in one spouse was a significant risk factor (odds ratio 10.0; 95% confidence interval 1.5-68.7; P=0.005) for oral HR HPV persistence in the other partner. Cumulative incidence of new HR HPV infections was identical in both spouses, while men seemed to clear their infection more rapidly. In univariate survival analysis, the partner's oral or genital HPV status, oral sex habits or age did not predict clearance or acquisition of oral HR HPV.
CONCLUSION:
Natural history of HPV infection in oral mucosa mimics that of genital HPV infection. Oral sex had no association to oral HPV infection, but a persistent oral HPV infection of the spouse increased the risk of persistent oral HPV infection 10-fold in the other spouse.
PMID:
16112613
[PubMed - indexed for MEDLINE]
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