No. The reason is steroids is a listed possible cause of hypothyroidism. If you are interested in lowering your thyroid antibodies then you need to lower your oxidative stress levels. Many studies show selenium lowers TPOAb levels. I personally had my TPOAb drop 80 IU eating brazil nuts (high in selenium). That wasn't too bad a drop but I did have other oxidative stress issues as well at the time - notably magnesium deficiency. When I was hyperthyroid my TPOAb rose from 470 IU to 1900 IU. Oxidative stress central!
*Medscape's article Selenium and the Thyroid Gland
Clin Endocrinol. 2013;78(2):155-164....
"Most authors attribute the effect of supplementation on the immune system to the regulation of the production of reactive oxygen species and their metabolites.
In patients with Hashimoto's disease and in pregnant women with anti-TPO antibodies, selenium supplementation decreases anti-thyroid antibody levels and improves the ultrasound structure of the thyroid gland. Although clinical applications still need to be defined for Hashimoto's disease, they are very interesting for pregnant women given that supplementation significantly decreases the percentage of postpartum thyroiditis and definitive hypothyroidism."
Excerpt from the article "What thyroid patients should know about Oxidative Stress"...
"Some Causes of oxidative stress
There are quite a few situations mentioned in articles and studies which can cause your body to be overly stressed from the results of oxidation and all the reactive oxygen species. They include, but are not limited to:
excess endurance exercising
excess weight lifting
lack of key antioxidant nutrients like Vitamin C, Vitamin E, Selenium, Magnesium and other minerals
excess radiation or sunlight
smoking (huge cause of oxidative stress)
excessive drinking or drug use
over-exposure to toxins in our air, water and foods like pesticides, chemicals, heavy metals and more
processed foods with all their artificial dyes, additives or flavorings
excess physical trauma
Graves disease aka hyperthyroidism
excess copper levels from the MTHFR defect"