I agree with Gimel.
Yes it is extremely common for TSH to be suppressed when taking medication with T3 in it (Cytomel) and even if taking sufficient amounts of T4 medication to relieve symptoms of Hypo.
The fact that the Dr was not or is not aware that T3 can cause suppression of TSH is an indication to me that he/she doesn't know how to effectively treat Hypo.
you are only hyper if you have symptoms of hyper. And as stated, your blood labs would other than TSH indicate if anything Hypo, NOT Hyper.
What are the symptoms you are having? What is your resting heart rate?
Also Osteoporosis is more a function of lack of testosterone than anything. even in women! plenty of studies that show getting testosterone levels back up to the age of a 29 year old reverses bone loss. 29 is the age that they use as a baseline for bone density. Unfortunately, "they" do not use 29 year old sex hormone levels to correspond to healthy, rather they are "age related" at best. And just because everyone has low testosterone at increased age, does not mean that it is healthy, only normal.
If every one has osteoporosis does that make it "normal". And if so would you really want to be "normal?" Or would you rather be healthy?
Therefore I'd recommend that you have your sex hormones tested (Estradiol, testosterone, progesterone, SHBG, and DHEA-S) if for no other reason than to establish a baseline for future reference.
Your Endo has no clue. TSH is a pituitary hormone that is affected by so many things that only at extreme values, in the untreated state, does it have any correlation with your thyroid status. Your thyroid status is best defined by the amount of T3 Effect in tissues throughout the body, which is dependent on the supply of, and response to, thyroid hormone. In turn, the supply of thyroid hormone is represented by serum Free T4 and Free T3 levels. TSH has only a weak correlation with Free T4 and Free T3, and a negligible correlation with Tissue T3 Effects. In addition there are other variables such as Vitamin D, cortisol and ferritin that affect the response to thyroid hormone at the tissue level.
It is also important to know that there are studies that have shown that many patients taking thyroid hormone at a dosage adequate to relieve their hypothyroid symptoms will have suppressed TSH levels. This is a result of taking a full dose of thyroid hormone once daily and the fact that T4 med establishes an equilibrium that is quite different from that with the usual continuous low flow of natural thyroid hormone in the untreated state. a suppressed TSH is a treated person does not mean hyperthyroidism unless there are attendant hyper symptoms due to excessive serum levels of Free T4 and Free T3. So TSH should not be used to determine thyroid hormone dosage.
The concern about low TSH and osteopenia is unnecessary. Note this quote from an excellent thyroid doctor. "Thyroid hormone replacement does not cause bone loss as is commonly believed; it simply increases all metabolic activities in the body. If a person is already in a bone-losing state, such as a postmenopausal woman who is not on proper bioidentical hormone replacement therapy, then she will lose bone faster with better thyroid levels". The solution to that is not to withhold necessary thyroid hormone, but to fix the underlying causes for the bone loss.
Excessive thyroid hormone levels can affect some heart issues; however, as explained above a low TSH does not indicate excess FT4 and FT3 in treated patients. Your results clearly show that. Even with the suppressed TSH, your Free T4 and Free T3 are not even up to mid-range, so why is your TSH to be accepted as an infallible indicator rather than your actual thyroid hormone levels?
Also, hypothyroid patients are frequently deficient in Vitamin D, B12 and ferritin. So you should be tested for those and then supplement as needed to optimize. D should be at least 50 ng/mL, B12 in the upper end of its range, and ferritin should be at least 100.
If you have any questions about any of this, please click on my name and then scroll down to my journal. At the end of the journal you will find a one page overview of a paper that I highly recommend reading, at least the first two pages, and more if you want to get into the discussion and scientific evidence for all that is stated here. Also, in the full paper see recommendation 13 on page 13 about suppressed TSH.