A respected ophthalmologist last week recommended ILM peel to remove residual ERM (from a botched November 2013 vitrectomy with ERM peel) that's causing severe macula pucker in my right (OD) eye.
I'm extremely hesitant to go through with the procedure. I'm worried about developing scotomas due to the peeling process. I've spent dozens of hours scouring this and other eye forums about ILM peeling and the results of same.
I've not found a whole lot of material on the subject. What I did find was distressing. In many instances people developed scotomas after ILM peeling. Several persons said their OCT showed cells in the IS/OS region were wiped out. One said the missing area looked like it was 'rubbed out with an eraser'. This person has a scotoma to the right of their central vision.
I've read a fair amount (10+) of fairly recent (2009 to 2016) studies about ILM peeling versus ERM only peeling. Virtually every study concludes ILM peeling prevents recurrence of ERM. But the studies disagree as to whether or not ILM peeling results in better BCVA as opposed to ERM peeling alone. The studies also disagree as to whether or not the risks of ILM peel outweigh its results (e.g. 100% prevention ERM recurrence). A 2014 study I read a few days ago concluded ILM peeling needs more study to determine long term benefits/detriments!
Most worrisome, practically every study says you can't help removing some of the neural layer when doing an ILM peel. The ILM is only 2 (maybe) microns thick. Despite a surgeon's expertise and experience, grasp cite areas seem to show the most damage of all (according to the last study I read). Overall, photoreceptor and Mueller cells seem subject to being removed; and the remaining cells of each damaged to some extent.
** This is exactly what worries me more than any other aspect of having an ILM peel. **
If there's the slightest chance of scotomas from the recommended ILM peel, I'd rather not have the peel and live with what I've got left after the first botched surgery.
It's not a question of faith in the surgeon who advised ILM peel. It's a question of "What if I get a scotoma, then what!?"
Nobody's perfect. Anyone can have a bad day at work. When an eye surgeon has a bad day at work the patient pays the ultimate price, not the surgeon. It's just the way it is. I've already paid the price once, in 2013. Hate to pay it again.
I'm a very outdoors person. In the next year or two I hope to visit Alaska, Yellowstone Park (extended back country trekking and camping), Glacier Park (MT), Colorado's 10th Mountain Hut system, and Idaho's Hell's Canyon. I can't imagine visiting these places and not being able to fully see and thus enjoy them because of scotomas.
I'd rather keep my slightly blurry 20/50, and my slight binocular diplopia (that makes me depressed sometimes)....rather than gain 20/40 (or better) with scotomas.
I've devoured everything I could find by JodiJ on this and other boards. Like everyone else, I'm extremely comforted by her research and unfailing contributions to us who stumble around in our search for answers. Even so, after about a month of research I've still not found a large enough body of posts to answer my question "How big or small is the risk of scotomas due to ILM peel?"
When I asked the surgeon about scotomas from ILM peel he said "They'll go away". I was so stunned at this answer that I didn't stop him right there and demand a more precise answer. Before I knew it we were onto the next subject. I'm very disappointed in myself for not asking more scotoma questions. To be fair, the surgeon's answer might be absolutely correct; and thus make absolute sense to him. But to me, in my ignorance, a scotoma indicates an retinal injury that will more than likely never self resolve.
*** Am I correct in that assessment? ***
The surgeon's office is 1,200 miles from where I live. Going back for more answers requires another flight, hotel, and rental car. Until I make those arrangements I'm trying to question those who've had ILM peel about their outcomes.
I thank everyone for reading and responding.